-
- Molecular Formula: C₆₂H₉₈N₁₆O₂₂
- Molecular Weight: 1419.55 g/mol
- Sequence: L-Valine,glycyl-L-alpha-glutamyl-L-prolyl-L-prolyl-Lprolylglycyl-L-lysyl-L-prolyl-L-alanyl-L-alpha-aspartyl-L-alpha-aspartyl-L-alanylglycyl-L-leucyl-;glycyl-L-alpha-glutamyl-L-prolyl-L-prolyl-L-prolylglycyllysyl-L-prolyl-L-alanyl-L- alpha-aspartylL-alpha-aspartyl-L-alanylglycyl-L-leucyl-L-valine
-
- Molecular Formula: C₂₁₂H₃₅₀N₅₆O₇₈S
- Molecular Weight: 4963 g/mol
- Sequence: Ac-Ser-Asp-Lys-Pro-Asp-Met-Ala-Glu-lle-GluLys-Phe-Asp-Lys-Ser-Lys-Leu-Lys-LysThr-Glu-Thr-Gin-Glu-Lys-Asn-Pro-Leu-Pro-Ser-Lys-GluThy-lleGlu-Gin-Glu-Lys-Gin-Ala-Gly-Glu-Ser
-
- Axin Regulation and Accelerated Cellular Migration: Successful tissue repair relies on the precise recruitment and execution of fibroblasts and immune cells to the site of cellular stress, where fibroblasts promote extracellular matrix formation. The baseline movement of these cells is highly dependent on the scaffold protein axin. Preclinical research suggests that the BPC-157 & TB-500 blend synergizes at the gene and structural level to maximize cell motility. BPC-157 promotes the transcription of the axin gene to increase protein expression and upregulate internal actin production. Concurrently, TB-500 acts as an actin-binding protein, sequestering actin in localized areas where cell movement is required, optimizing the formation of vital axin filaments.
- Soft-Tissue Remodeling and Skeletal Regeneration: Beyond cellular motility, both peptides are heavily scrutinized for structural muscle, tendon, and ligament regeneration. Studies indicate that elevated levels of TB-500 exhibit a direct stimulatory action on myoblasts and myocytes. This action is associated with reduced inflammation at injury sites, allowing researchers to observe accelerated skeletal muscle regeneration following trauma.
- Pro-Angiogenic Activation and Ischemic Repair: TB-500 is extensively advanced in preclinical cardiovascular models for its proposed involvement in cardiac muscle repair. In models simulating myocardial infarction, the peptide has been observed to reverse tissue damage by activating an endogenous response that triggers new blood vessel growth and restores perfusion to injured myocardium.
- Cystic Fibrosis and Sputum Cohesivity Kinetics: Outside traditional structural repair, the unique fluid-modulating traits of the Thymosin Beta-4 fragment make it an active candidate in respiratory research. Preclinical evaluation suggests that exposure to TB-500 alters rheological profiles, causing a distinct decrease in sputum cohesivity, which provides researchers with a stable framework for investigating cellular mechanics in cystic fibrosis models.
-
- Verified Purity: ≥ 99% purity guaranteed through mass spectrometry and HPLC profiling.
- USA Vetted: Independent laboratory confirmation ensures absolute consistency, correct peptide ratios, and complete freedom from residual chemical cross-contaminants.
- Research Use Only: This compound is synthesized exclusively for in vitro laboratory experimentation and preclinical evaluation. Make sure to double-check the physical label to confirm this information.




Reviews
There are no reviews yet.